TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Did Not Attend
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Poor
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Fair
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Good
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Very Good
TMG 202 Molecular diagnosis of PIK3CA-related overgrowth spectrum (PROS) in 162 patients and recommenda Excellent
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Did Not Attend
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Poor
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Fair
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Good
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Very Good
TMG 210 Delineating the impact of rare copy number variation on IQ in schizophrenia Excellent
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Did Not Attend
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Poor
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Fair
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Good
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Very Good
TMG 212 Exome reanalysis: Integration of new knowledge in genomic reports Excellent
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Did Not Attend
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Poor
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Fair
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Good
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Very Good
TMG 222 Investigation of simplex patients born to consanguineous parents: Whole exome sequencing has a high yield, with significant contribution of de novo mutations Excellent
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Did Not Attend
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Poor
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Fair
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Good
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Very Good
TMG 217 Homozygous mutation in RSPRY1 in a patient of Canadian indigenous descent results in skeletal dysplasia and intellectual disability: Expanding the phenotype of SEMD Faden-Alkuraya type Excellent